By Prof. Dr. med. Alfred Christian Feller, Professor emeritus Jacques Diebold (auth.)
During the ten years because the final variation of Histopathology of Non-Hodgkin's Lymphomas, in response to the up-to-date Kiel class, our wisdom on malignant lymphomas, particularly on extranodal lymphomas, has elevated. This quantity - Histopathology of Nodal and Extranodal Non-Hodgkin's Lymphomas - is an accelerated and fully revised variation, now in line with the WHO type. The parallels to the up to date Kiel category and the true class are indicated. the data is prepared in organ-specific chapters, comprising the well known nodal lymphoma entities in addition to all identified extranodal lymphomas and the several organ-specific clinico-pathological entities: lymphomas of the spleen, the gastrointestinal tract, the surface, and so on. as well as the morphology, the foremost immunohistochemical, molecular genetic, and medical information are incorporated in each one chapter.
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Extra info for Histopathology of Nodal and Extranodal Non-Hodgkin’s Lymphomas
26. Lymph node infiltrate of a primary nodal plasmacytoma with mostly mature, only slightly polymorphic plasma cells disease and is not pronounced. In the bone marrow there is a massive increase in the number of fibers and infiltration by small lymphoid cells; the latter eventually replacing the whole of the normal marrow. Substantial enlargement of mediastinal and retroperitoneal lymph nodes has been reported (Vardiman and Golomb 1984; Malik et al. 1989). The peripheral blood 41 42 4 Nodal B-Cell Lymphoma contains a small to moderate number of hairy cells.
Their cytoplasm is less basophilic and less abundant than that of plasma cells of the lymphoplasmacytic immunocytoma (Fig. 15). Frequently, the number of monoclonal plasmacytoid cells is much better seen in immunostainings, since these cells can easily be overlooked by conventional Giemsa stain (Fig. 16a, b). Furthermore, there are always a few immunoblasts. In some cases there are a larger number of immunoblasts, and there may be very large proliferation centers, as in tumor-forming B-CLL. Nevertheless, lymphocytes are still the predominant cells.
The cells are mostly paraimmunoblasts (Giemsa stain) Fig. 11. Immunohistochemical staining of the same tumor as in Fig. 8; with strong surface and intracytoplasmic IgM expression in a transformed B-CLL (immunoperoxidase stain) Richter syndrome; Figs. 9). In contrast, a terminallymphoblastic phase is evidently rare in CLL. 10 shows a case that was interpreted as the blastic phase of B-CLL. The "blasts" contained a large amount of cytoplasmic IgM and were CD23-negative (Fig. 11); instead, the cells looked more like prolymphocytes than immunoblasts.
Histopathology of Nodal and Extranodal Non-Hodgkin’s Lymphomas by Prof. Dr. med. Alfred Christian Feller, Professor emeritus Jacques Diebold (auth.)