By Kimber Jest
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Additional resources for Clinical Toxicology - Vol 48, issue 7, Aug 2010
Kim D, Tolliver TJ, Huang S, Martin BJ, Andrews AM, Wichems C, Holmes A, Lesch KP, Murphy DL. Altered serotonin synthesis, turnover and dynamic regulation in multiple brain regions of mice lacking the serotonin transporter. Neuropharmacology 2005; 49:798–810. 80. Fabre V, Beaufour C, Evrard A, Rioux A, Hanoun N, Lesch KP, Murphy DL, Lanfumey L, Hamon M, Martres MP. Altered expression and functions of serotonia 5-HT1A and 5-HT1B receptors in knock-out mice lacking the 5-HT transporter. Eur J Neurosci 2000; 12:2299–2310.
Hyman SE. A 28 year old man addicted to cocaine. J Am Med Assoc 2001; 286:2586–2594. 2. Berke JD, Hyman SE. Addiction, dopamine, and the molecular mechanisms of memory. Neuron 2000; 25:515–532. 3. Nestler EJ. Molecular basis of long-term plasticity underlying addiction. Nat Rev Neurosci 2001; 2:119–128. 4. Koob GF, LeMoal M. Drug abuse: hedonic homeostatic dysregulation. Science 1997; 278:52–58. 5. Hyman SE, Malenka RC. Addiction and the brain: the neurobiology of compulsion and its persistence.
0001). To further compare the efficacy of these two agents, the cyanide concentration was increased to 801 ppm; all Cbitreated animals survived, whereas only 60% of Na2S2O3treated animals survived (p = ns). However, at this cyanide concentration, the combination of Na2S2O3 and NaNO2, a recommended clinical treatment, was fully effective. To Clinical Toxicology vol. 48 no. 3 Fig. 4. Dose–response to cobinamide and OHC in the intraperitoneal model. 24 mmol/kg of KCN. Data were plotted as dose–response curves and the ED50 calculated by log transformation of the dose and nonlinear regression analysis of the data.
Clinical Toxicology - Vol 48, issue 7, Aug 2010 by Kimber Jest